Neuroplasticity: New biochemical mechanisms by Jean-Paul Macher, Jean-Pierre Olié

By Jean-Paul Macher, Jean-Pierre Olié

New study into neuroplasticity is an engaging new examine perspective which can bring about advancements in remedy for the depressive issues. during this quantity, researchers within the box give a contribution chapters reading various facets of this phenomenon. Figures and tables support clarify complex innovations. This ebook presents a brand new perspective at the therapy of depressive problems.

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PSQIPMPHZ Restoration of neuroplasticity  Impairments in neuroplasticity Glutamate levels, % change from baseline  Tianeptine Amygdala Figure 2. PSHBO$"
***"OYJPMZUJD FGGFDUTPGBOPWFMHSPVQ**NFUBCPUSPQJDHMVUBNBUFSFDFQUPS BHPOJTU -:  JO UIF GFBSQPUFOUJBUFE TUBSUMF QBSBEJHNJOIVNBOT1TZDIPQIBSNBDPMPHZ #FSM   REGULATION OF CELLULAR PLASTICIT Y IN MOOD DISORDERS: THE ROLE OF THE AMPA RECEPTOR Per Svenningsson and Bruce S. 1"SFDFQUPS  Saline-treated animals  Phosphorylated Ser845-GluR1 (% of saline level) Phosphorylated Ser 831-GluR1 (% of saline level) Acute    Cortex  Tianeptine-treated animals    Hippocampus Hippocampus CA3 CA1 Cortex Hippocampus CA1 Saline-treated animals  Phosphorylated Ser845-GluR1 (% of saline level) Phosphorylated Ser831-Glur1 (% of saline level) Chronic Hippocampus CA3    Cortex Hippocampus CA3 Hippocampus CA1  Tianeptine-treated animals    Cortex Hippocampus Hippocampus CA1 CA3 Figure 1.

1"SFDFQUPS  CELLULAR PLASTICIT Y AND THE PATHOPHYSIOLOGY OF DEPRESSION Thérèse M. FDIBOJTNT Neuromorphological changes in animal models of depression and in depressed patients Animal models Prefrontal cortex t3FEVDUJPOPGUIFOVNCFSBOEMFOHUIPGBQJDBMEFOESJUJDCSBODIFTJO UIFNFEJBMQSFGSPOUBMDPSUFY t3FEVDUJPO JO UIF WPMVNF PG UIF VQQFS QSFGSPOUBM MBZFST PG NFEJBM QSFGSPOUBMDPSUFY Hippocampus t%FOESJUJDSFNPEFMJOHJO$"QZSBNJEBMOFVSPOT
XJUISFWFSTJCMFBUSPQIZ TIPXOCZTIPSUFOJOHBOEEFCSBODIJOHPGBQJDBMEFOESJUFT t%FDSFBTFEWPMVNF t%FDSFBTFEMFWFMPG/BDFUZMBTQBSUBUF t%FDSFBTFEQSPMJGFSBUJPOSBUFPGHSBOVMFQSFDVSTPSDFMMT t%FDSFBTFEBEVMUOFVSPHFOFTJTJOUIFEFOUBUFHZSVT Amygdala t*ODSFBTFEEFOESJUJDBSCPSJ[BUJPOJOTQJOZQZSBNJEBMBOETUFMMBUFOFVSPOT Patients with chronic depression Prefrontal cortex t3FEVDFEWPMVNFPGHSFZBOEXIJUFNBUUFS t3FEVDFECSBJOPYZHFOBUJPO t3FEVDFEOFVSPOBMTJ[FBOESFEVDFEEFOTJUZPGHMJBMDFMMT Hippocampus t3FEVDFEWPMVNF CZVQUP t"MUFSBUJPOTJOCMPPEGMPXBOEHMVDPTFNFUBCPMJTN Amygdala t7PMVNF PG DPSF OVDMFJ EFDSFBTFE JO DISPOJD EFQSFTTJPO CVU UPUBM volume retained t5PUBMWPMVNFJODSFBTFEJOBGJSTUFQJTPEFPGEFQSFTTJPO t"MUFSBUJPOTJOCMPPEGMPXBOEHMVDPTFNFUBCPMJTN Orbitofrontal cortex t7PMVNFSFEVDFE Table 1.

PSQIPMPHZ Restoration of neuroplasticity  Impairments in neuroplasticity Glutamate levels, % change from baseline  Tianeptine Amygdala Figure 2. PSHBO$"
***"OYJPMZUJD FGGFDUTPGBOPWFMHSPVQ**NFUBCPUSPQJDHMVUBNBUFSFDFQUPS BHPOJTU -:  JO UIF GFBSQPUFOUJBUFE TUBSUMF QBSBEJHNJOIVNBOT1TZDIPQIBSNBDPMPHZ #FSM   REGULATION OF CELLULAR PLASTICIT Y IN MOOD DISORDERS: THE ROLE OF THE AMPA RECEPTOR Per Svenningsson and Bruce S. 1"SFDFQUPS  Saline-treated animals  Phosphorylated Ser845-GluR1 (% of saline level) Phosphorylated Ser 831-GluR1 (% of saline level) Acute    Cortex  Tianeptine-treated animals    Hippocampus Hippocampus CA3 CA1 Cortex Hippocampus CA1 Saline-treated animals  Phosphorylated Ser845-GluR1 (% of saline level) Phosphorylated Ser831-Glur1 (% of saline level) Chronic Hippocampus CA3    Cortex Hippocampus CA3 Hippocampus CA1  Tianeptine-treated animals    Cortex Hippocampus Hippocampus CA1 CA3 Figure 1.

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